Harris, Neil


Professor In-Residence, Neurosurgery

Member, Brain Research Institute

Cell & Developmental Biology GPB Home Area

Neuroscience GPB Home Area

Contact Information

Lab Number: 310-825-8646
Work Phone Number: 310-206-5691

Mailing Address:

Department of Neurosurgery
300 Stein Plaza
Los Angles, CA 90095


Professor Harris directs NEIL lab with over 25 years of experience with rodent CNS injury models and in particular using MRI and PET to assess structure and function. He received his B.Sc. in Biology/Neuroscience from University of Portsmouth in 1988, and his Ph.D. in Physiology from King’s College London in 1991. Dr. Harris’s early focus of research addressed the question of optimal timing for intervention after the diagnosis of infantile hydrocephalus. Prior to joining University of California Los Angeles (UCLA), Dr. Harris received training in multimodality imaging techniques, including PET, structural MRI, fMRI, DTI, and Glucose/blood-flow autoradiography at Kings college University of London, University of Florida McKnight Brain Institute, the Royal College of Surgeons unit of Biophysics in the Institute of Child Health, and University of Cambridge Department of Neurosurgery. Subsequently, Dr. Harris conducted studies to address forebrain ischemic stroke looking at the potential use of non-invasive biomarkers to determine salvageable areas of brain. The studies were cited amongst primary reported findings on biophysical mechanism of the change in water diffusion after stroke. Dr. Harris currently resides as Professor in Residence of UCLA Department of Neurosurgery where he primarily conducts investigations on Traumatic Brain Injury and is the scientific director of UCLA 7T animal imaging core.


A selected list of publications:

Shijo Katsunori, Sutton Richard L, Ghavim Sima S, Harris Neil G, Bartnik-Olson Brenda L   Metabolic fate of glucose in rats with traumatic brain injury and pyruvate or glucose treatments: A NMR spectroscopy study Neurochemistry international, 2017; 102: 66-78.
Sta Maria Naomi Sulit, Reger Maxine Lane, Cai Yan, Baquing Mary Anne Tezheart Mercado, Buen Floyd, Ponnaluri Aditya, Hovda David, Harris Neil G, Giza Christopher C   D-cycloserine restores experience-dependent neuroplasticity after TBI in the developing rat brain Journal of neurotrauma, 2016; 102: .
Molet Jenny, Maras Pamela M, Kinney-Lang Eli, Harris Neil G, Rashid Faisal, Ivy Autumn S, Solodkin Ana, Obenaus Andre, Baram Tallie Z   MRI uncovers disrupted hippocampal microstructure that underlies memory impairments after early-life adversity Hippocampus, 2016; 26(12): 1618-1632.
Brown Robert J, Jun Brandon J, Cushman Jesse D, Nguyen Christine, Beighley Adam H, Blanchard Johnny, Iwamoto Kei, Schaue Dorthe, Harris Neil G, Jentsch James D, Bluml Stefan, McBride William H   Changes in Imaging and Cognition in Juvenile Rats After Whole-Brain Irradiation International journal of radiation oncology, biology, physics, 2016; 96(2): 470-8.
Moro Nobuhiro, Ghavim Sima S, Harris Neil G, Hovda David A, Sutton Richard L   Pyruvate treatment attenuates cerebral metabolic depression and neuronal loss after experimental traumatic brain injury Brain research, 2016; 1642(2): 270-7.
Harris N G, Verley D R, Gutman B A, Sutton R L   Bi-directional changes in fractional anisotropy after experiment TBI: Disorganization and reorganization? NeuroImage, 2016; 133(2): 129-43.
Harris N G, Verley D R, Gutman B A, Thompson P M, Yeh H J, Brown J A   Disconnection and hyper-connectivity underlie reorganization after TBI: A rodent functional connectomic analysis Experimental neurology, 2016; 277(2): 124-38.
Shijo Katsunori, Ghavim Sima, Harris Neil G, Hovda David A, Sutton Richard L   Glucose administration after traumatic brain injury exerts some benefits and no adverse effects on behavioral and histological outcomes Brain research, 2015; 1614(2): 94-104.
Thomsen Gretchen M, Le Belle Janel E, Harnisch Jessica A, Mc Donald Whitney S, Hovda David A, Sofroniew Michael V, Kornblum Harley I, Harris Neil G   Traumatic brain injury reveals novel cell lineage relationships within the subventricular zone Stem cell research, 2014; 13(1): 48-60.
Moro Nobuhiro, Ghavim Sima, Harris Neil G, Hovda David A, Sutton Richard L   Glucose administration after traumatic brain injury improves cerebral metabolism and reduces secondary neuronal injury Brain research, 2013; 1535(1): 124-36.
Bartnik-Olson Brenda L, Harris Neil G, Shijo Katsunori, Sutton Richard L   Insights into the metabolic response to traumatic brain injury as revealed by (13)C NMR spectroscopy Frontiers in neuroenergetics, 2013; 5(1): 8.
Harris Neil G, Nogueira Marcia S M, Verley Derek R, Sutton Richard L   Chondroitinase enhances cortical map plasticity and increases functionally active sprouting axons after brain injury Journal of neurotrauma, 2013; 30(14): 1257-69.
Harris Neil G, Chen Szu-Fu, Pickard John D   Cortical reorganization after experimental traumatic brain injury: a functional autoradiography study Journal of neurotrauma, 2013; 30(13): 1137-46.
Harris Neil G, Mironova Yevgeniya A, Chen Szu-Fu, Richards Hugh K, Pickard John D   Preventing flow-metabolism uncoupling acutely reduces axonal injury after traumatic brain injury Journal of neurotrauma, 2012; 29(7): 1469-82.
Deng-Bryant Ying, Prins Mayumi L, Hovda David A, Harris Neil G   Ketogenic diet prevents alterations in brain metabolism in young but not adult rats after traumatic brain injury Journal of neurotrauma, 2011; 28(9): 1813-25.
Harris Neil G, Mironova Yevgeniya A, Hovda David A, Sutton Richard L   Chondroitinase ABC enhances pericontusion axonal sprouting but does not confer robust improvements in behavioral recovery Journal of neurotrauma, 2010; 27(11): 1971-82.
Harris Neil G, Mironova Yevgeniya A, Hovda David A, Sutton Richard L   Pericontusion axon sprouting is spatially and temporally consistent with a growth-permissive environment after traumatic brain injury Journal of neuropathology and experimental neurology, 2010; 69(2): 139-54.
Giza Christopher C, Kolb Bryan, Harris Neil G, Asarnow Robert F, Prins Mayumi L   Hitting a moving target: Basic mechanisms of recovery from acquired developmental brain injury Developmental neurorehabilitation, 2009; 12(5): 255-68.
Harris N G, Carmichael S T, Hovda D A, Sutton R L   Traumatic brain injury results in disparate regions of chondroitin sulfate proteoglycan expression that are temporally limited Journal of neuroscience research, 2009; 87(13): 2937-50.